U.S. study charts changing prevalence of profound and non-profound autism

Out-of-focus photograph of a young girl sitting at a desk using an eraser.

Community traits: Children with profound autism are more likely to be girls than are their peers with non-profound autism.

Laurence Mouton / Getty Images

More than a quarter of autistic 8-year-olds in the United States have profound autism, according to a study published today in Public Health Reports. Although the overall prevalence of profound autism in this age group increased from 2002 to 2016, the prevalence of non-profound autism did so at a sharper rate, the findings show.

The results align with the idea that much of autism’s dramatic rise over the past three decades stems from diagnostic changes. “We are now finding people with autism who don’t have intellectual disability and who have more fluent language” — and who may not have been diagnosed 20 or 30 years ago, says Catherine Lord, distinguished professor of psychiatry and education at the University of California, Los Angeles, who was not involved in the work.

The term profound autism, introduced in December 2021 by Lord and her colleagues as part of the Lancet Commission on the Future of Care and Clinical Research in Autism, describes autistic people who likely need lifelong 24/7 care, many of whom have intellectual disability, limited communication abilities or both. The commission estimated at the time that anywhere from 18 to 48 percent of the autistic population might fit this description, according to an analysis of three separate datasets from the U.S., the United Kingdom and Norway.

The new estimate — the first based on data from 20,135 autistic children, collected by the U.S. Centers for Disease Control and Prevention’s Autism and Developmental Disabilities Monitoring (ADDM) Network — falls in the middle of that range at 26.7 percent.

But non-white autistic children are more likely than their white peers to have profound autism, the work shows. The prevalence is 76 percent higher for Black children, 55 percent higher for children from Asian, Native Hawaiian or other Pacific Islander backgrounds, 50 percent higher for Hispanic children and 33 percent higher for American Indian (Native American) and Alaska Native children. Children with profound autism are also more likely to come from low socioeconomic backgrounds compared with their peers with non-profound autism.

Those differences point to disparities in how clinicians diagnose autism across different communities, Lord says.

“We probably are not picking up the more able kids with autism in minority ethnic groups,” she says. “It tells us more about who’s not there than who’s there.”

Profound autism prevalence also varies by gender and region, the study shows. The results can help researchers and clinicians plan for the services and supports that people who meet the criteria for profound autism may need over their lifetime, says study investigator Michelle Hughes, an epidemiologist at the CDC. “We saw this as an opportunity to bring data to the conversation,” she says.

The ADDM Network has released reports on autism prevalence at study sites in the U.S. every two years since 2000. For those reports, the researchers use data from medical and educational records to estimate the number of 4- and 8-year-olds with autism in a given year. The network is continuing to track autism prevalence at its current sites and announced yesterday that it plans to expand its surveillance to 5 additional sites — including one in Puerto Rico — for the next funding cycle.

In the new study, CDC researchers used a similar approach to estimate the number of 8-year-olds with profound autism across 15 sites for the surveillance years between 2000 and 2016, excluding 2012 and 2014 because they did not have information on children’s verbal abilities for those years. They classified children as having profound autism if a clinician reviewing their records determined they fit the diagnostic criteria for autism and their records indicated that they were nonverbal, minimally verbal or had an IQ below 50.

In 2000, about 27 of every 10,000 children within the network had profound autism, and 39 in 10,000 had non-profound autism, the team found. And although prevalence increased for both groups over the next 16 years, that of non-profound autism did so more rapidly — reaching 143 per 10,000, compared with 46 per 10,000 for profound autism.

Group change: As more children with non-profound autism are diagnosed over time, the proportion of the autistic population with profound autism decreases.

Courtesy Hughes et al. / CDC

Profound autism prevalence is also relatively stable across regions, whereas the prevalence of children with non-profound autism varies more widely, the study shows: from 260 per 10,000 in New Jersey to 104 per 10,000 in Colorado for the 2016 surveillance year.

Those regional differences may be explained by how accessible services are in a given state, as well as the site’s racial and ethnic makeup, Lord says.

Autistic girls are about 25 percent more likely to be classified as having profound autism than their male peers, the findings show. Although it is possible that many autistic girls with non-profound autism are not diagnosed, it may also be that girls are more likely than boys to survive with the genetic mutations that cause this presentation of autism, Lord says.

The findings fit, to a large degree, with what the Lancet commission expected, Lord says. For example, most of the children with profound autism have low adaptive-functioning scores, the new study shows, and they are more likely than children with non-profound autism to have self-injurious behaviors and seizures. Having a more complete picture of who these children are and what traits they have will help researchers and clinicians provide better recommendations for their well-being, she says.

But the study is the first of its kind and needs to be replicated, Lord adds, noting methodological challenges such as incomplete records. “The numbers could be high — or low,” she says, because the researchers had to make inferences on data that was not systematically collected.

Moving forward, it will also be important to gain a better understanding of autism in the non-white population, says Santhosh Girirajan, associate professor of genomics at Pennsylvania State University in University Park, who was not involved in the work.

“What we use in white populations might not be applicable in non-white populations,” Girirajansays. Addressing that might take redefining some of the assessments to account for cultural and familial differences, he says — and may ultimately help the field better understand autism as a whole.

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A questionable study linked epidurals to autism. Then what?

Image of an epidural being administered.

Scientific reflection: A seemingly simple epidemiology paper prompted researchers to consider what publishing is for.

The spread of misinformation — and the way fear can burn through a landscape — was forefront in Tor-Arne Hegvik’s mind when, in October 2020, he opened up his web browser and found a new autism paper in JAMA Pediatrics. Hegvik is a gynecologist and researcher at the University of Bergen in Norway, and the study, co-led by biostatistician Anny Xiang of Kaiser Permanente Southern California (KPSC), made what appeared to be an improbable claim: The epidural analgesia given during labor is associated with increased chances of having a child with autism.

Hegvik had never wondered whether epidurals could contribute to autism, and he didn’t think anyone else had seriously considered it either. He assumed the association detailed in the paper was due to confounders — unaccounted-for factors that skew a result. Plus, he had clinical duties to attend to and patients to see. He tried to put it out of his mind.

But the paper continued to plague him. It reminded him of Andrew Wakefield’s infamous fraudulent statement that vaccines cause autism: Though the link was easy to disprove, it was exceedingly difficult to quash in the public consciousness. The epidural paper was not exactly that — Hegvik could see it was honest science — but the potential for harm felt similar. The decision to get an epidural requires clear, straightforward information. The Xiang paper, he worried, would muddy the waters. He couldn’t turn away. So he emailed some of his colleagues and asked: Would anyone be interested in trying to refute this?

Other worried researchers were also talking about the paper, united by the thought that the study’s implied conclusion — that epidurals cause autism — could become a Wakefield-esque fiasco. It is difficult to know exactly what fears will catch fire in the public consciousness, but if it was even a possibility here, they wanted to do something about it.

The first person at JAMA Pediatrics to read the epidural paper was the journal’s chief editor, Dimitri Christakis. Christakis rejects on sight about three-quarters of the papers he receives. The rest go to his team of associate editors, some then go on to peer review, and in the end just 6 percent of submissions are accepted. So it’s safe to say JAMA Pediatrics is a discerning publication. Today, Christakis doesn’t remember anything particularly noteworthy about Xiang’s paper or its peer-review process. (The journal does not make its reviews public.)

What he saw was a study showing that among nearly 148,000 babies born in a KPSC hospital between 2008 and 2015, children whose mothers had an epidural while giving birth were 37 percent more likely to be autistic than those whose mothers did not have one; the longer the epidural exposure, the higher the chance of autism. Christakis felt there were some weaknesses in the data, but the researchers acknowledged them and were cautious in their conclusions. The methodology was not flawed on its face. He felt the question Xiang had introduced deserved an answer.

Christakis’ decision to publish revolved largely around the role of scientific publishing. Part of the job of a journal, he told me, is to present ideas for its community to evaluate. He could find no reason not to present Xiang’s. JAMA Pediatrics accepted the paper on 17 June 2020 and published it on 12 October.

If the Xiang paper presented an intriguing scientific idea, it did not appear to be one the research community had previously cared much about. According to PubMed, before 2020 just one study had ever specifically looked at epidurals and autism together — a 2004 Australian paper that found a small association in about 2,000 people.

“I thought, when it first came out, ‘Oh my goodness, this is just going to permeate every discussion I have for the rest of my career.’” Daniel McIsaac

In their paper, Xiang and her colleagues were careful not to state that epidurals might cause autism. They had simply observed an association and noted that it should be viewed “with caution” and “cannot be interpreted as a demonstration of a causal link.”

But Hegvik thought the cautioning statements did not go far enough.

“When you read the article, they never directly claim that epidurals might cause autism,” Hegvik said. “But it’s like, everywhere you read between the lines, that’s basically what they’re writing.”

Daniel McIsaac, an anesthesiologist at Ottawa Hospital and associate professor of anesthesiology at the University of Ottawa in Canada, felt much the same after reading the Xiang paper. “I thought, when it first came out, ‘Oh my goodness, this is just going to permeate every discussion I have for the rest of my career,’” he said.

McIsaac, like many others, was concerned that the study failed to account for factors that could make a person more likely to both use an epidural and give birth to an autistic child. Xiang and her colleagues, for example, did not control for the mother’s own psychiatric conditions, even though such conditions in parents are widely known to be linked to autism in children. Xiang and her team also excluded women who had received cesarean sections, which often occur because of complications with labor.

What’s more, it’s hard to see how epidurals — which come at the end of a pregnancy and generally last just a few hours — could lead to autism, a condition thought to originate in the earliest stages of fetal development. Because of this, McIsaac couldn’t understand why the research was even done. Typically, to justify doing a major epidemiological study like this one, a researcher needs good reason to think there’s something to find. “Just because you have data,” McIsaac said, “is never the right reason to do a study.”

Beyond citing some preliminary research in people, Xiang and her team also looked at a 1998 study comparing 11 infant rhesus monkeys born to mothers that had received a dose of bupivacaine (one of the drugs in a standard epidural) with eight control rhesus infants. The study showed that “standard clinical doses” of anesthetic drugs can “alter normal behavioral development,” according to Xiang’s paper. But that 1998 study concluded that even though the time course of the exposed rhesus infants’ development was shifted, the drug “does not cause neonatal abnormalities or specific cognitive deficits,” the researchers wrote.

Alexander Butwick, professor of anesthesiology at Stanford University in California, found the rhesus monkey data referenced in Xiang’s paper to be “pretty weak,” he told me. He and a colleague commented on the JAMA Pediatrics website that the dose used in the animal study wasn’t comparable to the one used in the delivery ward and called the researchers’ interpretations of relevant studies in people “at best misleading and at worst erroneous.”

Butwick set out to repeat the Xiang study with fresh data, and to do it quickly. “We really felt like we had a responsibility here to do not just the work but also respond in a timely manner,” Butwick said. “We were worried that the longer this was out there, the more people may feel like there’s a genuine cause-and-effect issue going on.”

He called epidemiologist Elizabeth Wall-Wieler, then assistant professor of community health sciences at the University of Manitoba in Canada (she now works in California), who had access to a dataset that linked clinical data from hospital visits to parent socioeconomic information and long-term outcomes for the child. Butwick and Wall-Wieler pulled numbers on 123,175 babies born in Manitoba from 2005 through 2016, adjusted for more than 30 factors — including maternal anxiety and depression — and found that epidurals were not associated with autism. That finding stood in a secondary analysis of siblings born to the same mother.

The paper was published 19 April 2021, also in JAMA Pediatrics. It was the first time the journal had published conflicting results on the same topic within 12 months — such a rare occurrence that Christakis pointed it out in an editor’s note published the same day.

Still, the papers around epidurals and autism kept coming. JAMA published two on the same day, in late September 2021. One, which looked at nearly 400,000 births in British Columbia, Canada, found a small association, though the authors ascribed it to “residual confounding.” The other, in nearly half a million children from a Danish registry, found no link at all. In a commentary accompanying the two papers, Cynthia Wong, chair of the Department of Anesthesia at the University of Iowa, wrote that “current evidence does not justify” considering the chances of autism when making decisions about epidurals.

In December 2021, the British Journal of Anesthesia published yet another null result, this time in almost 625,000 children born in Denmark. It was closely followed by a commentary from Wong and Butwick in the same journal, asserting “no meaningful association” between epidurals and autism. The case, they argued, was closed.

But four months later, JAMA Network Open published a study involving more than 650,000 mother-child pairs in Ontario, Canada, that found a small link between epidurals and autism — though the research team, which included McIsaac, noted the finding “must be interpreted with caution.” There were two meta-analyses, both of which found little reason to continue investigating the question. In fact, in the two and a half years since Xiang’s paper came out, there have been at least 28 papers addressing the purported connection. If the research community had not been interested in a link between autism and epidurals before Xiang’s study, it certainly was now. (Xiang declined to be interviewed by phone for this story, but in an email sent via a publicist affiliated with Kaiser Permanente, she repeated that her paper had only noted an association and added that she and her team welcomed additional studies. Xiang also stressed the benefits of epidurals for mothers and noted that there is not enough evidence to recommend against them.)

There was one more paper coming. Amir Sariaslan, a psychiatric epidemiologist at the University of Oxford in the U.K., was on the receiving end of Hegvik’s October 2020 email about Xiang’s study. He had never studied epidurals and had limited experience studying autism, but he had worked with Hegvik before and, crucially, could tap national registry data from Finland and Sweden, which contained information on more than 3.5 million births. Hegvik had access to data from another million births in Norway, and together they pulled together a game-changing cohort: 4.5 million people born across 28 years, with more than 1 million of them delivered using an epidural.

It took two years to finish the study; the findings were published online in the American Journal of Obstetrics and Gynecology last August. In a crude analysis, Sariaslan and his team found that children exposed to epidurals were 12 percent more likely to be autistic and 20 percent more likely to have attention-deficit/hyperactivity disorder than their unexposed counterparts.

Other studies had made initial steps beyond these preliminary associations. But because of the size of the cohort, Sariaslan had the power to do a new kind of analysis. He and his team pared their sample down to include only full siblings — other studies had confirmed just a shared mother. They then pulled just those siblings who differed both in their exposure to epidurals and whether or not they had autism or ADHD.

That specific group was about 2 percent of the original 4.5 million. But it was still 94,000 people. That was sufficient to find that people with either condition were no more likely than their siblings without a neurodevelopmental condition to have been exposed to an epidural.

In other words, the association between autism and epidurals completely disappeared.

“I personally think we’re done with this,” Wong said after Sariaslan’s study came out. There would be nothing to gain from mining more databases or a larger dataset. “There’s no place to go.”

The point of scientific publishing is to go “wherever science leads,” Christakis told me. Much of the pushback after the Xiang paper was published, he said, was by people worried that “this is going to panic women” about epidurals. “But that’s not a reason to not publish science. It’s just not.”

Science works only when researchers are free to publish their findings, regardless of what societal structures they may upend or whom they might upset. That march toward truth relies on the free flow of information. But the human brain is not always so rational. When encountering a new concept, people tend to focus on and remember the parts that cause negative emotions, like fear and anger. When that happens, collective efforts to debunk new ideas can sometimes just reinforce them.

“We were worried that the longer this was out there, the more people may feel like there’s a genuine cause and effect issue going on.” Alexander Butwick

That could have happened with Xiang’s work. The paper alarmed not only scientists but also doctors, and on the initial day of publication, five U.S. medical societies representing more than 100,000 physicians posted a response saying the study “does not provide credible scientific evidence” for the association and urging women not to be fearful of epidurals. By the end of that month, the Royal College of Anaesthetists, the Royal Australian and New Zealand College of Obstetricians and Gynaecologists and the Canadian Anesthesiologists’ Society had also all pushed back against the paper’s findings.

Their intention was much the same as Hegvik’s: to quickly refute what they saw as a potentially harmful conclusion in a paper based on science that could be challenged. But Dominique Brossard, an expert in the communication of science and risk at the University of Wisconsin-Madison, told me that rushing out a press release was, in her mind, exactly what not to do. The collective statement from physicians on the day of the publication, and those in the days that followed, could have stirred up a long-lasting media buzz — the kind that leaves the public recalling a shadowy link between epidurals and autism, and not much else.

Yet it did not. As far as anyone could tell me, the sweeping fear over epidurals hasn’t materialized in the general population. No one could cite more than a handful of instances from the past three years of a pregnant person asking about the link between autism and epidurals; most said they had never been asked about it. Searches for the topic turned up little on social media. It’s possible this is because the press was otherwise occupied in October 2020: It was furiously covering the global pandemic, and the United States was a month away from a contentious presidential election. Major national outlets such as The New York Times, The Washington Post and The Los Angeles Times, as well as popular science news sites such as Scientific American and New Scientist, did not cover Xiang’s study. (Spectrum ran a story the day after the study came out.)

That’s one theory. But Brossard has another. And that is that there was no organized movement eagerly waiting to pounce on new data about epidurals, no group that could use it to further a specific viewpoint. There is no anti-epidural lobbying group, for instance, and there is no epidural-alternative product to sell. If there had been people seeking to amplify Xiang’s study for personal gain, maybe it would have caught fire in the public consciousness.

Instead, though, the issue fizzled. That just left the scientists, gathering their evidence, publishing the story it told.

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Every April Autism Speaks celebrates World Autism Month, beginning with the United Nations-sanctioned World Autism Awareness Day on April 2. This year marks the 16th annual World Autism Awareness Day.

Throughout the month, we focus on sharing stories and providing opportunities to increase understanding and acceptance of people with autism, fostering worldwide support. This year, we are committed to standing together to make a world of difference for people on the spectrum.

Supporters can participate in World Autism Month in a variety of ways:

  • Pledge your support and uploading your photo to become part of our worldwide mosaic of unity and collaboration.
  • Make a meaningful, lasting impact for people with autism with your gift. It’s one of the easiest and best ways to show your support. Donate today!
  • Join our 2.8 million social followers to help foster understanding and acceptance by sharing the diverse stories of people on the spectrum or telling your own. Submit your story for our social media pages.
  • Engage in the Autism Speaks Kindness Campaign to fundraise for the autism community while sharing purposeful acts of kindness that encourage acceptance, understanding and inclusion.
  • Learn all the ways you can fundraise on social media.
  • Show your support by wearing our official World Autism Month gear.
  • Light It Up Blue on April 2 in celebration of people with autism and those who love and support them. Join the thousands of landmarks, buildings, homes and communities around the world and come together on April 2, World Autism Awareness Day to shine a light on the autism community.

Whether you have autism, love someone who does or are looking to support a diverse, accepting and kind community – let’s stand together and pledge our support to make a world of difference by helping all people with autism reach their full potential.

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Queremos visibilizar y concienciar sobre tres aspectos esenciales que contribuirán a promover la participación y la igualdad de oportunidades de las personas autistas y sus familias.

  • VARIABILIDAD dentro del espectro del autismo.Iguales, pero diferentes. 
  • SINGULARIDAD y ESPECIFICIDADDerribar estigmas y falsas creencias. Comprender el autismo y valorar a las personas autistas.  
  • SENTIDO DE PERTENENCIAOrgullo frente a rechazo; apoyo frente a reemplazo.  

Si quieres ser parte del cambio y ayudar a construir una sociedad más respetuosa con las personas con autismo en las que se respeten estos principios, te animamos a firmar el manifiesto.

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José Ramón Alonso, doctor en Biología de la Universidad de Salamanca explica los avances en investigación, las nuevas estrategias de diagnóstico y tratamiento sobre autismo, que según descubrimientos recientes está afectando a una gran cantidad de población que hasta hoy se desconocía.

Explica José Ramón que son muchos los esfuerzos que se están realizando para descubrir sus posibles causas pero que también se le está dando importancia al aspecto social, en apoyo a quienes la padecen y familias afectadas.

“Autismo es un trastorno del desarrollo y por lo que sabemos se inicia antes del nacimiento”. También, menciona múltiples equivocaciones relacionadas con teorías que han tratado de explicar sus orígenes, así como el uso de instrumentos que se están utilizando para su detección

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Explicando la evolución del “autismo” como diagnóstico

¿Por qué el autismo fue inicialmente considerado una condición psiquiátrica?

Cuando Leo Kanner, un psiquiatra y médico austríaco-americano, describió por primera vez el autismo en 1943, escribió sobre niños con “soledad autista extrema”, “ecolalia retardada” y un “deseo ansiosamente obsesivo de mantener la misma”. También señaló que los niños eran a menudo inteligentes y algunos tenían una memoria extraordinaria.

Como resultado, Kanner vio el autismo como una profunda perturbación emocional que no afecta a la cognición. De acuerdo con su perspectiva, la segunda edición del DSM, el DSM-II, publicado en 1952, definió el autismo como una condición psiquiátrica, una forma de esquizofrenia infantil marcada por un desapego de la realidad. Durante los años 50 y 60, se pensaba que el autismo tenía sus raíces en las madres frías y poco emotivas, a las que Bruno Bettelheim apodó “madres de la nevera”.

¿Cuándo se reconoció el autismo como un trastorno del desarrollo?

El concepto de “madre refrigeradora” fue refutado en los años sesenta y setenta, ya que un creciente número de investigaciones demostró que el autismo tiene fundamentos biológicos y está arraigado en el desarrollo del cerebro. El DSM-III, publicado en 1980, estableció el autismo como su propio diagnóstico independiente y lo describió como un “trastorno generalizado del desarrollo” distinto de la esquizofrenia.

Las versiones anteriores del manual dejaban muchos aspectos del proceso de diagnóstico abiertos a las observaciones e interpretaciones de los médicos, pero el DSM-III enumeraba los criterios específicos necesarios para un diagnóstico. Definía tres características esenciales del autismo: falta de interés en las personas, graves deficiencias de comunicación y respuestas extrañas al entorno, todas ellas desarrolladas en los primeros 30 meses de vida.

¿Cuánto tiempo duró esta definición?

El DSM-III fue revisado en 1987, alterando significativamente los criterios del autismo. Amplió el concepto de autismo añadiendo un diagnóstico en el extremo suave del espectro, trastorno generalizado del desarrollo no especificado de otra manera (PDD-NOS) y eliminando el requisito de aparición antes de los 30 meses.

Aunque en el manual no se utilizaba la palabra “espectro”, el cambio reflejaba la creciente comprensión entre los investigadores de que el autismo no es una condición única, sino un espectro de condiciones que pueden presentarse a lo largo de la vida.

El manual actualizado enumeraba 16 criterios en los tres dominios previamente establecidos, 8 de los cuales debían cumplirse para un diagnóstico. La adición de PDD-NOS permitió a los clínicos incluir a los niños que no cumplían completamente con los criterios de autismo, pero que aún requerían apoyo en el desarrollo o en el comportamiento.

¿Cuándo se presentó por primera vez el autismo como un espectro de condiciones?

El DSM-IV, publicado en 1994 y revisado en el 2000, fue la primera edición que clasificó el autismo como un espectro.

Esta versión enumeraba cinco condiciones con características distintas. Además del autismo y el PDD-NOS, añadió el “trastorno de Asperger”, también en el extremo leve del espectro; el “trastorno desintegrador de la infancia”, caracterizado por graves retrocesos y regresiones en el desarrollo; y el síndrome de Rett, que afecta al movimiento y la comunicación, principalmente en las niñas. El desglose se hizo eco de la hipótesis de la investigación en el momento en que, el autismo, tiene sus raíces en la genética, y que cada categoría se vincularía en última instancia a un conjunto de problemas y tratamientos específicos.

¿Por qué el DSM-5 adoptó la idea de un espectro continuo?

A lo largo de los años 90, los investigadores esperaban identificar los genes que contribuyen al autismo. Después de que el Proyecto del Genoma Humano se completó en 2003, muchos estudios trataron de concentrarse en una lista de “genes del autismo”. Encontraron cientos, pero no pudieron relacionar ninguno exclusivamente con el autismo. Se hizo evidente que no sería posible encontrar fundamentos genéticos y los tratamientos correspondientes para las cinco condiciones especificadas en el DSM-IV. Los expertos decidieron que lo mejor sería caracterizar el autismo como un diagnóstico integral, que fuera de leve a severo.

Al mismo tiempo, existía una creciente preocupación por la falta de coherencia en la forma en que los médicos de los distintos estados y clínicas llegaban a un diagnóstico de autismo, síndrome de Asperger o PDD-NOS. El aumento de la prevalencia del autismo en el decenio del 2000 sugirió que los médicos, a veces, se dejaban influir por los padres que presionaban para obtener un determinado diagnóstico o se veían influidos por los servicios disponibles en su estado.

Para abordar ambas preocupaciones, el DSM-5 introdujo el término “trastorno del espectro autista”. Este diagnóstico se caracteriza por dos grupos de características: “deterioro persistente en la comunicación e interacción social recíproca” y “patrones de comportamiento restringidos y repetitivos”, ambos presentes en la primera infancia. Cada grupo incluye conductas específicas, un cierto número de las cuáles los clínicos tienen que identificar. El manual eliminó el síndrome de Asperger, el PDD-NOS y el autismo clásico, pero debutó con un diagnóstico de trastorno de comunicación social, para incluir a los niños que sólo tienen impedimentos de lenguaje y sociales. El trastorno desintegrador de la infancia y el síndrome de Rett fueron eliminados de la categoría de autismo.

¿Por qué el DSM-5 generó tanta preocupación y controversia?

Incluso antes de que el manual se publicara en 2013, muchas personas con autismo y sus cuidadores se preocupaban por su efecto en sus vidas. A muchos les preocupaba que después de que su diagnóstico desapareciera del libro, perdieran los servicios o la cobertura del seguro. Aquéllos que se identificaron con el síndrome de Asperger, dijeron que el diagnóstico les dio un sentido de pertenencia y una explicación para sus desafíos; temían que quitar el diagnóstico fuera sinónimo de perder su identidad. Y los expertos no estaban de acuerdo en si los criterios de diagnóstico más estrictos del DSM-5 bloquearían los servicios para aquéllos con rasgos más leves o frenarían adecuadamente las tasas de prevalencia de la enfermedad.

Cinco años más tarde, está claro que el DSM-5 no cortó los servicios para las personas ya diagnosticadas con una condición de espectro autista. Sin embargo, un creciente conjunto de pruebas demuestra que sus criterios sí excluyen a más personas con rasgos más leves, niñas e individuos mayores que el DSM-IV.

¿Hay alternativas al DSM?

Los médicos de muchos países, incluido el Reino Unido, utilizan la Clasificación Internacional de Enfermedades (CIE). Publicada en la década de 1990, la edición actual y la 10ª de ese manual, agrupa el autismo, el síndrome de Asperger, el síndrome de Rett, la DDC y el PDD-NOS en una sola sección de “Trastornos Generalizados del Desarrollo”, tal como lo hacía el DSM-IV.

¿Cómo se ve el futuro para el diagnóstico del autismo?

Los expertos continúan viendo el autismo como un espectro continuo de condiciones. No hay revisiones planeadas para el DSM por ahora, pero el lenguaje en un borrador del ICD-11, que se estrenó en mayo de 2018, refleja los criterios del DSM-5. En el ICD-11, los criterios de autismo se trasladan a una nueva sección dedicada a los “Trastornos del Espectro de Autismo”.

El ICD-11 difiere del DSM-5 en varios aspectos clave. En lugar de requerir un número o combinación de características para un diagnóstico, lista las características de identificación y permite a los clínicos decidir si los rasgos de un individuo coinciden. Debido a que el DCI está destinado a un uso global, también establece criterios más amplios y menos específicos culturalmente que el DSM-5. Por ejemplo, pone menos énfasis en los juegos a los que juegan los niños, que en si siguen o imponen reglas estrictas a esos juegos. El DCI-11 también hace una distinción entre el autismo con y sin discapacidad intelectual, y destaca el hecho de que las personas mayores y las mujeres a veces enmascaran sus rasgos autistas.

Publicado en AUTISMO, DEFINICIONES | 26 comentarios


Un separatista echa a su hijo con asperger por hablar en español: “Si España está por encima de la familia, márchate”

Le quería “catalanizar”, asegura el joven muy afectado. Ahora está “en situación de desamparo”, explica su abogada, Vanessa González.

“En Cataluña no hay libertad”, asegura Juan -nombre ficticio-. Es la conclusión de un joven de 21 años, diagnosticado de Síndrome de Asperger cuando tenía tres, al que su propio padre ha dejado sin casa y sin trabajo sencillamente por hablar en español, sin importarle la situación de desamparo en la que se quedaba. “Es independentista radical”, explica el chico en declaraciones a Libertad Digital.

Está “muy disgustado”. “No pensaba que era tan mala persona”, comenta. Ellos discutían a menudo por sus diferencias políticas, pero no creyó que el hombre pudiese llegar tan lejos.

Llevaba desde 2018 trabajando en una de sus empresas -aunque “sin contrato”- y se fue a vivir con él hace diez meses, cuando a su madre la desahuciaron del piso en el que vivía.

Es decir, su padre era conocedor de la paupérrima situación económica de la mujer y de que ésta no podría hacerse cargo del joven si le echaba.

Aún así, lo hizo. “Si España está por encima de la familia, márchate”, le espetó. Ipso facto se quedó sin nada. No le dejó llevarse sus pertenencias. 

Ni siquiera su medicación, nos asegura. Juan no entiende la posición de su padre. “No respeta la lengua castellana, que según la ley y el Estatuto de Autonomía de Cataluña se ha de respetar”, indica.

Él tiene muy claro que está en su derecho de hablar como le plazca.

Prohibido hablar español

España es mi tierra… Mi país, mi patria. Me siento español y vivo en una tierra que es territorio nacional.

Yo hablo en castellano, pienso en castellano y nadie me tiene que obligar hablar en catalán”, afirma con contundencia.

No respetan mi españolidad, en Cataluña no hay libertad”, insiste.

“Aquí hay gente radical que quiere imponerte lo suyo. Pero España es un país muy plural, con riqueza de idiomas y riquezas culturales”.

La discusión que desembocó -el pasado viernes- en esta situación, la de que le echara de casa y del trabajo, empezó de la forma más tonta. Como otras tantas veces. Iban juntos, caminando por la calle a comprar el pan, cuando su padre le advirtió de que le iba a “catalanizar”.

Le exigió “que no hablase en castellano, que lo tenía prohibido” y le amenazó con echarle si lo hacía.

Esta disputa ya la habían tenido en otras ocasiones, su padre estaba empeñado en que “le tenía que hablar a los clientes en catalán”.

Y no es que nos sepa, añade su abogada Vanessa González, es que “se siente más cómodo hablando en español”. Otro de sus empeños era que no tuviese relación con su madre, de ideas políticas muy diferentes a las suyas (nada nacionalistas).


Publicado en NOTICIAS | 136 comentarios


New Treatment is Changing the Lives of Children, Adults and Families Impacted by Autism

The NeuroHealth Center – bringing Personalized Repetitive Transcranial Magnetic Stimulation to autistic patients in New Jersey – is now open in Hackettstown

The NeuroHealth Center specializing in Personalized Repetitive Transcranial Magnetic Stimulation (PrTMS®) – a pain-free, drug-free, noninvasive procedure that can change the life of children and adults with Autism Spectrum Disorder (ASD) – is now open in Hackettstown, New Jersey. The center is inspired by Frank and Mary Plut, who witnessed the impact PrTMS had on their young son, Frankie, and are passionate about helping other families affected by ASD.

Autism Spectrum Disorder (ASD) “When Frankie was eight months old, we knew something wasn’t right,” Frank shares. “At 18 months old, he was diagnosed with autism. Nonverbal – with multiple daily meltdowns and dangerous behaviors – we tried everything.”

Desperate to help their child, Frank and Mary learned about PrTMS, a life-changing therapy that gave them hope … and results.

“We took Frankie to see Kevin T. Murphy, MD, inventor of PrTMS, in California, the only location administering this unique therapy at the time,” Frank explains. “Frankie went through a series of daily weekday treatments for several weeks.”

Specially designed to meet the individual needs of each patient, PrTMS gently delivers personalized, repetitive magnetic pulses to five or six targeted areas of the brain. During each treatment session, an electromagnetic coil is placed near the patient’s scalp at these targeted locations on the head. The electromagnetic field painlessly stimulates the brain’s neurons, rebuilding the abnormal cranial minicolumns in ASD patients and improving the brain’s executive functioning.

Each treatment takes approximately 20 minutes. Most patients are treated five times a week – Monday through Friday – for six to 12 weeks. An EEG is taken every five days to determine progress and the treatment’s path forward. Within the first two weeks, it can be determined if the patient is responding to the therapy. If the patient responds positively, continued treatment is recommended. If the patient is not responding in the first two weeks, the recommendation will be to discontinue treatment.

Thanks to PrTMS, today Frankie is a mainstreamed 8th grade student.

“Frankie is completely conversant,” Frank continues. “He reads and writes, is social, no longer has meltdowns, narrates his own YouTube channel … and no longer toe walks.”

“The difference between the child we took to California and the one we returned home with was night and day. On the plane to California, Frankie was just making baby babble. On the way back, he was looking out the window saying ‘Goodbye California! See you soon.’ This was a kid who was nonverbal 30 days prior to the treatment, and now he is speaking and his feet are flat on the ground rather than walking on his toes.”

In addition to helping children like Frankie by potentially transforming non-verbal patients into verbal children, reducing meltdowns and diminishing toe walking, PrTMS may change patients’ lives by:

  • Reducing destructive behaviors
  • Promoting social interactions and eye contact
  • Fostering adaptability to smoothly transition from one activity to another
  • Decreasing anxiety and enhancing stress coping mechanisms
  • Improving overall mood and reducing depression
  • Promoting sleep
  • Advancing memory and mental clarity
  • Increasing attention span and ability to focus

Each child’s brain is unique, and the 30-day treatment window varies with each individual.

PrTMS is FDA approved to treat Major Depressive Disorder and may be used Off Label (not covered by insurance) to treat autism, anxiety disorder, traumatic brain Injury, post-traumatic stress disorder, attention deficit hyperactive disorder, stroke, dementia, Parkinson’s, obsessive-compulsive disorder and other neurological disorders.

You can also learn more about the NeuroHealth Center and PrTMS at You may also email or call 973.453.5423.

Publicado en AUTISMO, NOTICIAS | 53 comentarios


Northwestern Investigators Develop New Therapy for Autism Subtype

A team of Northwestern investigators led by Peter Penzes, PhD, the Ruth and Evelyn Dunbar Professor of Psychiatry and Behavioral Sciences and director of the Center for Autism and Neurodevelopment, has developed a new therapy that could treat Phelan-McDermid syndrome, a subtype of autism spectrum disorder (ASD), according to findings published in Molecular Psychiatry.  

According to the Centers for Disease Control and Prevention, one in 44 children in the U.S. have been diagnosed with ASD and each patient presents diverse clinical and developmental symptoms, which may include delayed speech, motor skills and learning skills, epilepsy, poor eating and sleeping habits, and gastrointestinal issues.  

Phelan-McDermid syndrome is known to be caused by a specific genetic mutation in SHANK3, a well-known ASD candidate gene. Due to the heterogeneity of ASD, the development of effective targeted therapies has been extremely challenging, leaving patients with treatment options that improve disease management. 

Based on the urgent need for new therapies, Penzes’ team developed a derivative of an insulin-like growth factor-binding protein, IGFBP2, which is found in structures of the brain affected in ASD and has been shown to improve neuroplasticity and cognitive functions.  

In the current study, the investigators administered the IGFBP2-derived peptide, called JB2, to mice with SHANK3 mutations. Through advanced brain imaging, they found that the drug improved neuroplasticity, behavior impairments and cellular processes in the mices’ brains.  

These changes were directly correlated with improvements in the mices’ learning and memory skills, motor function and communication through ultrasonic vocalizations, findings that could be translated to social behaviors in humans, according to Penzes. 

“In patients with autism, speech doesn’t develop at all, or it develops very late, or it’s very simplified, so these ultrasonic vocalizations in mice are thought to somehow model that in mice,” said Penzes, who is also professor of Neuroscience and of Pharmacology

Using electroencephalography to measure the mice’s brain activity, the team also discovered that JB2 normalized neuronal excitability, or how neurons respond to stimuli with an electric charge, and seizure susceptibility.  

While the drug is still in early stages, Penzes said, theoretically, routinely administering the drug into patients’ bloodstreams while they are young either through regular injections or in pill form would be ideal.   

“Because this is a neurodevelopmental condition, the brain still changes after birth, even into adulthood. The earlier one could intervene, the better. So, it would be preferable to start it as a pediatric medication, but those are more difficult to get approval for,” Penzes said.   

Penzes noted that initial clinical trials would involve participants with Phelan-McDermid syndrome and if successful, could eventually expand to include patients with other types of ASD.  

“The assumption is that similar changes are happening in the brains of patients with other types of autism and in Phelan-McDermid syndrome, but these patients would have a better response,” Penzes said.  

Co-authors of the study include Sehyoun Yoon, PhD, research assistant professor of Neuroscience, Jeffrey Burgdorf, PhD, adjunct professor of Biomedical Engineering at the McCormick School of Engineering, Marc Dos Santos, PhD, a postdoctoral fellow in the Penzes laboratory, and Joseph Moskal, PhD, professor of Biomedical Engineering at the McCormick School of Engineering.  

This work, part of ongoing efforts by the Center for Autism and Neurodevelopment to develop new therapies, was supported by National Institute of Mental Health grant R41MH121140. 

FUENTE:Northwestern Investigators Develop New Therapy for Autism Subtype – News Center

Publicado en AUTISMO, INVESTIGACION | 34 comentarios


Piden una mayor protección para las personas con discapacidad ante los abusos de la banca

El Cermi ha reclamado que las personas afectadas pueden verse excluidas al ser consumidoras de las entidades financieras.

l Comité Español de Representantes de Personas con Discapacidad (CERMI) ha reclamado al Parlamento que proteja más eficazmente a los consumidores con discapacidad ante los abusos de las entidades financieras.

El CERMI ha elaborado un documento de propuestas de enmiendas al Proyecto de Ley por el que se crea la Autoridad Administrativa Independiente de Defensa del Cliente Financiero.

En el documento, reclama un protocolo de atención a personas con discapacidad y personas mayores para garantizar la inclusión financiera y la necesidad de protección reforzada de las personas con discapacidad como consumidoras de servicios financieros ante el mayor riesgo de exclusión motivado por la suma de factores asociados a la discapacidad.

El CERMI, que propone 38 enmiendas y modificaciones a las disposiciones adicionales y finales, valora que el actual proyecto de Ley «haya recogido parte de las alegaciones planteadas por la sociedad civil de la discapacidad en el proceso de audiencia pública» para favorecer su inclusión financiera y garantizar sus derechos como consumidores de servicios financieros. 

Publicado en AUTISMO, DIVULGACIÓN | 48 comentarios


La Academia Americana de Pediatría (AAP), los Centros para el Control de Enfermedades, los Institutos Nacionales de Salud, las asociaciones y programas para padres con niños con autismo y los investigadores han llamado la atención sobre la importancia de la detección precoz de los trastornos del espectro autista (TEA) y el acceso a la posible intervención temprana. La literatura científica indica que la edad media de diagnóstico de TEA es de 4 años, sin embargo, cerca de la mitad de los niños con este trastorno del desarrollo neurológico puede ser detectado a los 14 meses de edad. La Academia Americana de Pediatría recomienda que en las prácticas de Pediatría se utilicen los cuestionarios específicos para la detección de los signos de autismo al inicio de los 18 meses. Estos cuestionarios no son perfectos ya que, a menudo, requieren que los padres respondan “sí” o “no” a las preguntas acerca de la presencia de ciertos comportamientos de los niños. Las conductas de los niños asociadas con los trastornos del espectro autista (TEA) se presentan frecuentemente de una manera inconsistente, y se confunden con los comportamientos observados en niños con desarrollo típico o normal. Estas conductas observadas en los niños con los trastornos del espectro autista muchas veces difieren del desarrollo típico del niño, no solo en la frecuencia sino también en la calidad. Para mejorar el reconocimiento de los primeros signos de los trastornos del espectro autista, entre los pediatras, padres y terapeutas que participan de la detección temprana, la Dra. Rebecca Landa, una investigadora dedicada a los trastornos del espectro autista y la directora del Centro de Autismo y Trastornos Relacionados del Instituto Kennedy Krieger en Baltimore (Estados Unidos), ha desarrollado un tutorial gratuito de 9 minutos con las señales de comportamiento de niños con autismo de un año de edad. El tutorial consta de seis vídeo-clips que comparan niños pequeños que no muestran signos de TEA con los niños que muestran signos tempranos de TEA. Cada vídeo es presentado por una voz que describe y explica cómo los comportamientos específicos que presenta el niño, a medida que ocurren en la pantalla, son indicativos de TEA o del desarrollo típico de un niño. Los videos e información presentados en este tutorial, se obtuvieron a través de la investigación realizada por la Dra. Rebecca Landa, financiada por el Instituto Nacional de Salud Mental, y se centraron en indicadores tempranos de los trastornos del espectro autista y en las características del desarrollo temprano de niños con y sin autismo. Para obtener más información, por favor visite
Publicado en AUTISMO, INVESTIGACION | 71 comentarios


En el marco del Día Mundial de Concienciación sobre el Autismo, la Sociedad Británica de Autismo busca crear empatía por las personas que viven con este trastorno psicológico
Publicado en AUTISMO, INVESTIGACION | 55 comentarios

Thinking Across the Spectrum

Dr. Temple Grandin comes to Google to talk about her book: The Autistic Brain: Thinking Across the Spectrum.

When Temple Grandin was born in 1947, autism had only just been named.

Today it is more prevalent than ever, with one in 88 children diagnosed on the spectrum.

And our thinking about it has undergone a transformation in her lifetime: Autism studies have moved from the realm of psychology to neurology and genetics, and there is far more hope today than ever before thanks to groundbreaking new research into causes and treatments.

Now Temple Grandin reports from the forefront of autism science, bringing her singular perspective to a thrilling journey into the heart of the autism revolution.

Weaving her own experience with remarkable new discoveries, Grandin introduces the neuroimaging advances and genetic research that link brain science to behavior, even sharing her own brain scan to show us which anomalies might explain common symptoms.

We meet the scientists and self-advocates who are exploring innovative theories of what causes autism and how we can diagnose and best treat it. Grandin also highlights long-ignored sensory problems and the transformative effects we can have by treating autism symptom by symptom, rather than with an umbrella diagnosis.

Most exciting, she argues that raising and educating kids on the spectrum isn’t just a matter of focusing on their weaknesses; in the science that reveals their long-overlooked strengths she shows us new ways to foster their unique contributions.

From the “aspies” in Silicon Valley to the five-year-old without language, Grandin understands the true meaning of the word spectrum.

The Autistic Brain is essential reading from the most respected and beloved voices in the field.

Publicado en AUTISMO, INVESTIGACION | 27 comentarios



Publicado en DIVULGACIÓN | 40 comentarios


En esta charla basada en hechos, la genetista Wendy Chung comparte lo que sabemos sobre el trastorno del espectro autista. Por ejemplo, que el autismo tiene múltiples, tal vez causas entrelazadas. Más allá de la inquietud y la preocupación que puede rodear a un diagnóstico, Chung y su equipo miran a lo que hemos aprendido a través de estudios, tratamientos y la escucha atenta.
Publicado en AUTISMO, INVESTIGACION | 10 comentarios

Gene mutation

Gene mutation leading to autism found to overstimulate brain cells

Scientists looking to understand the fundamental brain mechanisms of autism spectrum disorder have found that a gene mutation known to be associated with the disorder causes an overstimulation of brain cells far greater than that seen in neuronal cells without the mutation.

The Rutgers-led study, spanning seven years, employed some of the most advanced approaches available in the scientific toolbox, including growing human brain cells from stem cells and transplanting them into mouse brains.

The work illustrates the potential of a new approach to studying brain disorders, scientists said.

Describing the study in the journal, Molecular Psychiatry, researchers reported a mutation — R451C in the gene Neurologin-3, known to cause autism in humans — was found to provoke a higher level of communication among a network of transplanted human brain cells in mouse brains. This overexcitation, quantified in experiments by the scientists, manifests itself as a burst of electrical activity more than double the level seen in brain cells without the mutation.

“We were surprised to find an enhancement, not a deficit,” said Zhiping Pang, an associate professor in the Department of Neuroscience and Cell Biology in the Child Health Institute of New Jersey at Rutgers Robert Wood Johnson Medical School and the senior author on the study. “This gain-of-function in those specific cells, revealed by our study, causes an imbalance among the brain’s neuronal network, disrupting the normal information flow.”

The interconnected mesh of cells that constitutes the human brain contains specialized “excitatory” cells that stimulate electrical activity, balanced by “inhibitory” brain cells that curtail electrical pulses, Pang said. The scientists found the oversized burst of electrical activity caused by the mutation threw the mouse brains out of kilter.

Autism spectrum disorder is a developmental disability caused by differences in the brain. About 1 in 44 children have been identified with the disorder, according to estimates from the Centers for Disease Control and Prevention.

Studies suggest autism could be a result of disruptions in normal brain growth very early in development, according to the National Institutes of Health’s National Institute of Neurological Disorders and Stroke. These disruptions may be the result of mutations in genes that control brain development and regulate how brain cells communicate with each other, according to the NIH.

“So much of the underlying mechanisms in autism are unknown, which hinders the development of effective therapeutics,” Pang said. “Using human neurons generated from human stem cells as a model system, we wanted to understand how and why a specific mutation causes autism in humans.”

Researchers employed CRISPR technology to alter the human stem cells’ genetic material to create a line of cells containing the mutation they wanted to study, and then derived human neuron cells carrying this mutation. CRISPR, an acronym for clustered regularly interspaced short palindromic repeats, is a unique gene-editing technology.

In the study, the human neuron cells that were generated, half with the mutation, half without, were then implanted in the brains of mice. From there, researchers measured and compared the electrical activity of specific neurons employing electrophysiology, a branch of physiology that studies the electrical properties of biological cells. Voltage changes or electrical current can be quantified on a variety of scales, depending on the dimensions of the object of study.

“Our findings suggest that the NLGN3 R451C mutation dramatically impacts excitatory synaptic transmission in human neurons, thereby triggering changes in overall network properties that may be related to mental disorders,” Pang said. “We view this as very important information for the field.”

Pang said he expects many of the techniques developed to conduct this experiment to be used in future scientific investigations into the basis of other brain disorders, such as schizophrenia.

“This study highlights the potential of using human neurons as a model system to study mental disorders and develop novel therapeutics,” he said.

Other Rutgers scientists involved in the study include Le Wang, a postdoctoral associate in Pang’s lab, and Vincent Mirabella, who is earning doctoral and medical degrees as part of the MD-PhD student at Robert Wood Johnson Medical School; Davide Comoletti, an assistant professor, Matteo Bernabucci, a postdoctoral fellow, Xiao Su, a doctoral student, and Ishnoor Singh, a graduate student, all of the Rutgers Child Health Institute of New Jersey; Ronald Hart, a professor, Peng Jiang and Kelvin Kwan, assistant professors, and Ranjie Xu and Azadeh Jadali, postdoctoral fellows, all of the Department of Cell Biology and Neuroscience, Rutgers School of Arts and Sciences.

Thomas C. Südhof, a 2013 Nobel laureate and professor in the Department of Molecular and Cellular Physiology at Stanford University, contributed to the study, as did scientists at Central South University in Changsha, China; SUNY Upstate Medical Center in Syracuse, N.Y.; University of Massachusetts in Amherst, Mass.; Shaanxi Normal University in Shaanxi, China; and Victoria University in Wellington, New Zealand.

Publicado en INVESTIGACION | 36 comentarios


El sufrimiento de un niño autista por la pirotecnia: le ponen cascos pero no para de llorar

El joven sufre “reiterados ataques de ansiedad todos los años a causa de los petardos”

La pirotecnia se suele utiliza de manera indiscriminada en fechas de celebración, como en Navidad, lo que puede causar daños a la salud, especialmente en las personas con trastorno del espectro autista (TEA).

En las últimas horas se ha hecho viral un vídeo que muestra el sufrimiento de un niño con TEA durante una cena navideña a causa de la pirotecnia, con el que el perfil en Twitter @Ireecabreraa ha querido “concienciar sobre el daño que produce en personas” que padecen este tipo de trastornos.

“Necesito 30 segundos de vuestra atención. Este es mi hermano Alejandro y esta es su realidad. Todos los años sufre reiterados ataques ansiedad debido a los petardos”, rezaba el tuit.

Tal y como señala la Federación Autismo Madrid, la hipersensibilidad de algunas personas con este tipo de trastornos hacen que reacciones muy intensamente a estímulos como el ruido, las luces o el dolor, llegando incluso a poder sentir un gran malestar y convirtiéndose el entorno en un lugar agresivo, altamente estresante y abrumados.

Por su parte, @ireecabreraa añade que “esto deriva a que no perciban los estímulos auditivos como nosotros, provocando este tipo de reacción ante ruidos de alto impacto”. Precisamente en este vídeo se aprecia cómo Alejandro, a pesar de tener los casos puestos, no puede dejar de manifestar ese malestar.

La publicación ha recibido miles de interacciones en las últimas horas y cuenta con casi dos millones de reproducciones.

Publicado en AUTISMO, NOTICIAS | 34 comentarios


Sin irnos por lo teórico y la enumeración de largas listas científicas sobre cómo detectar a un asperger, un investigador reconocido en el campo de los T.E.A.

Tony Atwwood, lo reduce a unas pocas “pistas” que podrán decirte sí esa persona con la que estudias, trabajas o simplemente tu amigo es asperger.

1. Un asperger suele ser literal entonces será esa persona que te comunica lo que siente y piensa, Es decir, no tiene filtro social.

2. Es muy perseverante en sus ideas y su perspectiva a pesar de las opiniones de otros por su inflexibilidad mental.

3. Es aquella persona que te hablará de sus intereses con detalle y entusiasmo es una enciclopedia sobre temas que sean de su gusto.

4. Notarás lo que Tony Attwood llama Habla “Aspergiana”, un lenguaje social caracterizado por una comunicación sin dobles sentidos, vocabulario avanzado y muy técnico.

5. Los verás centrados en los detalles, porque captan todo desde lo particular hacia lo general. . 6. Puedes ser ese compañero o compañera que recuerda, textos, libros, la carpeta donde se guardó hace dos años un documento, pero olvida citas y reuniones.

7. Es el ordenado, el rutinario, el cuadriculado. No es fácil que los veas cambiar su agenda o su rutina diaria.

8. Es el niño o compañero de trabajo sensible hacia sabores, olores, texturas y ambientes ruidosos.

9. Pueden ser también aquellos que se sientan solos a almorzar o que se junta con personas igual de solitarias o tan “raras” como ellos mismos.

10. Son personas que buscan hacer lo justo y lo correcto.

Publicado en AUTISMO, DEFINICIONES | 50 comentarios